Volume 3, Issue 1 (Spring 2000)
J Arak Uni Med Sci 2000, 3(1): 33-38 |
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Hashemieh M. The Study of Glucose-6-Phosphate Dehydrogenase Deficiency among icteric hospitalized neonates in Amir-Kabir and Taleghani Hospitals in Arak in 1998-1999. J Arak Uni Med Sci 2000; 3 (1) :33-38
URL: http://jams.arakmu.ac.ir/article-1-6611-en.html
URL: http://jams.arakmu.ac.ir/article-1-6611-en.html
School of Medicine, Arak University of Medical Sciences, Arak, Iran.
Abstract: (1378 Views)
The various enzymatic defects of red blood cells cause some genetical diseases, of which the most important and the most common is G6PD deficiency. G6Pd deficiency is one of the most common causes of hemolysis in newborn period and can lead to anemia, hyperbilirubinemia, and kernicterus. In this study we tries to find out incidence of glucose-6-phosphate dehydrogenase deficiency in icteric hospitalized neonates in newborns wards of Taleghani and Amir Kabir hospitals, in Arak city from December 1998 to January 1999. We concluded that: The total hospitalized, icteric neonates were 332, 63.86% were male and 36.14% of them were female. The icteric neonates with G6PD deficiency were 6.02% and 93.98% of them had normal G6Pd enzyme. All of the G6Pd deficient neonates were male. The onset of icter in G6Pd-deficient neonates was between second and 6th day. In 80% of icteric G6PD deficient neonates, the bilirubin total was over 18 mg/dl.G6Pd deficiency in neonates with hemolytic anemia were, 12.5%. Positive familiar history in icteric neonates with G6PD deficiency were 15%. The rate of exchange transfusion among icteric G6PD deficient patients were 15%. The most common blood group in icteric neonates with G6PD deficiency was B+ (incidence 50%)
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